Reduce Embarrassing (and Painful) Crohn’s and Irritable Bowel Disease Symptoms Naturally

IBS Natural Remedies

We’ve all had those “gotta go” moments…looking for a bathroom…afraid we may not make it in time.

But for the millions with Crohn’s disease—a chronic inflammatory bowel disease—that kind of thing can happen every day. And that’s not even the worst part. Crohn’s is not just embarrassing, but very painful. Symptoms can include abdominal cramping, fever, fatigue, skin and mouth ulcers, and diarrhea or constipation…among others.1

But there’s good news. And even if you don’t have Crohn’s it could help you too…

Researchers from Virginia Tech may have found natural relief.2 They conducted a study using 13 people with Crohn’s disease.

Participants took 6,000 mg of a specific nutrient daily for 12 weeks. Researchers found that the nutrient suppressed levels of specific T cells that cause inflammation.

Using the Crohn’s disease activity index (CDAI)—a way to measure disease symptoms—patients reported major improvements. CDAI dropped from 245 to 187. They also reported an increase in quality of life.

And even though this supplement was tested on patients with Crohn’s, you don’t have to suffer from the disease to reap the benefits. Many people suffer from similar irritable bowel symptoms. The cause?  Inflammation. Stop inflammation, stop the symptoms. Whether it’s from Crohn’s or any other bowel disorder.

What nutrient helped the patients… and relieved inflammation?

Conjugated linoleic acid (CLA).

“CLA represents a promising new supportive intervention for gut inflammation,” researchers from the Virginia Tech study said.

Doctors often prescribe several medications, including steroids, to treat symptoms. But they come with risky side effects. CLA on the other hand, was well tolerated and caused no side effects.

CLA is a type of fatty acid found in beef and dairy. Due to the rise in low-fat diets, people these days don’t usually get enough CLA.

Scientists have been studying CLA’s effects on immune function and inflammation for years.3 For example…

In another study out of Virginia, researchers tested CLA on mice.4 It helped reduce inflammation and colorectal cancer in the mice.

That’s great news for anyone plagued with irritable bowel symptoms. And not just because they’ll feel relief. Fact is, they’re more likely to develop certain types of bowel cancers.5

The cause of Crohn’s disease is unknown.6 Genetics, hereditary, and environmental factors may all play roles in gut inflammation. Diet and stress can aggravate the disease.

And while there is no “cure” for Crohn’s, remission is possible with the right lifestyle and diet changes. Upping your intake of CLA is a great way to start.

Raw milk and grass-fed, free range beef are rich in this nutrient. Though to hit the 6,000 mg the study used, you should supplement. CLA is easy to find at any local health food store. If you suffer from Crohn’s disease or any of the painful symptoms you could enjoy relief in less than 12 weeks.

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Health Topic: General Health | Pain Relief


  1. Nick P says:

    If this is true about CLA then what about this: PLEASE RESPOND TO COMMENT

    Prostaglandins Leukot Essent Fatty Acids. 2012 Oct-Nov;87(4-5):135-41. doi: 10.1016/j.plefa.2012.08.004. Epub 2012 Sep 5.

    Lowering dietary linoleic acid reduces bioactive oxidized linoleic acid metabolites in humans.

    Ramsden CE, Ringel A, Feldstein AE, Taha AY, MacIntosh BA, Hibbeln JR, Majchrzak-Hong SF, Faurot KR, Rapoport SI, Cheon Y, Chung YM, Berk M, Mann JD.

    Linoleic acid (LA) is the most abundant polyunsaturated fatty acid in human diets, a major component of human tissues, and the direct precursor to the bioactive oxidized LA metabolites (OXLAMs), 9- and 13 hydroxy-octadecadienoic acid (9- and 13-HODE) and 9- and 13-oxo-octadecadienoic acid (9- and 13-oxoODE). These four OXLAMs have been mechanistically linked to pathological conditions ranging from cardiovascular disease to chronic pain. Plasma OXLAMs, which are elevated in Alzheimer’s dementia and non-alcoholic steatohepatitis, have been proposed as biomarkers useful for indicating the presence and severity of both conditions. Because mammals lack the enzymatic machinery needed for de novo LA synthesis, the abundance of LA and OXLAMs in mammalian tissues may be modifiable via diet. To examine this issue in humans, we measured circulating LA and OXLAMs before and after a 12-week LA lowering dietary intervention in chronic headache patients. Lowering dietary LA significantly reduced the abundance of plasma OXLAMs, and reduced the LA content of multiple circulating lipid fractions that may serve as precursor pools for endogenous OXLAM synthesis. These results show that lowering dietary LA can reduce the synthesis and/or accumulation of oxidized LA derivatives that have been implicated in a variety of pathological conditions. Future studies evaluating the clinical implications of diet-induced OXLAM reductions are warranted.

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