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Clinical data
Trade namesCataflam, Voltaren, others [1]
Pregnancy categoryAU: C US: C (Risk not ruled out) in 1st and 2nd trimester, D in 3rd trimester
Routes of administrationBy mouth, rectal, intramuscular, intravenous (renal- and gallstones), topical
ATC codeD11AX18 ( WHO ) M01AB05 ( WHO ), M02AA15 ( WHO ), S01BC03 ( WHO )
Legal status
Legal statusAU: S2 (Pharmacy only) – S4, UK: POM (Prescription only) (P for topical formulation), ℞-only in most preparations/countries, limited OTC in some countries, manufacture and veterinary use is banned in India, Nepal, and Pakistan due to imminent extinction of local vultures
Pharmacokinetic data
Protein bindingMore than 99%
MetabolismLiver, oxidative, primarily by CYP2C9, also by CYP2C8, CYP3A4, as well as conjugative by glucuronidation (UGT2B7) and sulfation; [2] no active metabolites exist
Elimination half-life1.2–2 hr (35% of the drug enters enterohepatic recirculation)
Excretion40% biliary 60% urine

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Long-term administration of NSAIDs has resulted in how to use diclofenac gel papillary necrosis and other renal injury. Renal toxicity has also been seen in patients in whom renal prostaglandins have a compensatory role in the maintenance of renal perfusion.Call your doctor for medical advice about side effects.

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The decrease in sodium reabsorption is responsible for the natriuresis required to maintain adequate renal perfusion. At the distal nephron, PGI2 enhances potassium secretion through the renin-angiotensin system as it stimulates renin release.Case-control and cohort epidemiological studies showed that concomitant use of drugs that interfere with serotonin reuptake and an NSAID may potentiate the risk of bleeding more than an NSAID alone.

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